Julie, thanks for posting this. It’s helping build the knowledge on this. That copy of the email Dana gave you is probably the most I’ve read on this. With specifics about care at least.. She seems to be a go to person having first hand experience of CWP syndrome. I have to admit, I read thru this this morning before work and was left a bit saddened. It does help though to have some proactive measures to manage things
The thing that confuses me is the description of the poos for Cow poop bunnies. The large and soft or marshmellowy does not fit Jersey or Schroeder it seems. Though Dana did say in her reply to me “Sometimes the condition first manifests as a variety of poop shapes other than the normal “cocoa puff” type.”
The pic of Schroeders poos is what I get with Jersey all the time. She’ s been drinking alot more over the last 6 weeks or so but still pretty dry poop. I’m reposting this from other thread as it refers to sodium and I wonder if this has anything to do with the drier poops, though hear it is attributed to the mush poop. There’s also some things that I missed the first time round reading this that are cause for concern – such as “excessive thinness” and possible connection to E.C> (???)
2) Megacolon
The megacolon (congenital aganglionic) and ‘an inherited disorder of the GI tract, the more’ common gene in rabbits with En En, ie ‘what gives it the characteristics of the mantle Hotot, English Spot, etc.. ie, white with black or brown stains around the eyes and / or back.
This is an abnormal accumulation of feces in the intestines, which can not be expelled feces symptoms include huge, misshapen, often inflammation; you can have frequent episodes of diarrhea or loss of mucus alternating with moments of gut stasis. Bunnies with this problem have difficulty ‘to assimilate nutrients from food, sometimes not produce ciecotrofi and often have problems of excessive thinness.
It seems that this problem is caused by a malformation or malfunction of the colon and / or blind. It was’ discovered that rabbits with En En gene show a reduction in the rate of absorption of sodium through the wall of the cecum. This indicates a Excessive liquefaction of ingested material in the proximal part of the large intestine.
This might be a late complication resulting from (and amended by) various exogenous and endogenous sources of stress, worsened by the metabolic status of the rabbits En En ipotireotico but it ‘very probable that the genetic difference compared with rabbits with a single gene is precisely En at the intestine.
The only therapy is to administer prokinetic drugs and extra hydration during episodes of stasis and to follow the diet with particular attention, always providing much hay.
It ‘possible that the neurological symptoms (or stress) RESULTING FROM’ Encephalitozoonosis cuniculi cause intestinal malfunctions. source: translate.google.com.au/translate
Jersey has been losing weight. At first I thought it just a small difference with scales at the vet but now I’m seeing and feeling it too and have been thinking she could have intestinal worms or something. I’m feeling like a hypochondriac. Something for another thread perhaps.
Does Shroeder have weight issues/fluctuations? Also, do you ever see him produce cecals or eat his regular poop? Eating fecals is the first thing that brought me to finding out about CWP. Lightchick had posted about Lizzie doing this (excessively at the time) and since Jersey was doing this too, I did some searches. CWP came up because there was the thought that fecal and cecal matter were mixed.